So now you are saying that tobacco companies would not want to emphasize a positive benefit from using their products. Even if it is only a small benefit to a small part of the population. They are content with the ‘smoking causes death’ label on the package.
Even the pharmaceutical companies pull this shit, they want EVERYONE to take their drugs…
“Do you feel like today is a day? Have you been breathing? Have you been known to urinate occasionally after you drink fluids? If you answered yes to all these questions, we can make your life better with our drug!”
Smoking doesn’t always cause death. And yes, they’d rather have an image that can get everyone to smoke rather than shame people who we know aren’t diagnosed for nicotinic compounds… It’s called corporate strategy. Most people don’t even know nicotinic compounds help schizophrenia symptoms, if this was made public, people would be ashamed to smoke for fear of the label of schizophrenia, and it would shame people who didn’t have it to not smoke because they did’nt need it. Trust me, they’ve spent billions figuring all this out.
Your argument is that tobacco companies want everyone to smoke and therefore they will not claim a health benefit for schizophrenics. Instead, they are content to have their product seen as dangerous for everyone.
Not that it matters because I’m leaving ILP soon, but you said you’d move all my threads to off topic. This was a valid thread for government because it was in todays paper that they wanted to raise the age to 25. You just have a grudge against me. You brought your friends over to laugh at my understanding of suicidology when you were the one making the math mistake out of ignorance (males basically have a zero percent attempt rate) and you ended up with a number of 5:240 for females with your stupid math. You may have others convinced that you’re winning all these debates but not everyone.
It’s still an Ecmandu thread, which means it’s going to be a bunch of unsourced, fantasy-land bullshit in which you make preposterous claims based on no data, and other people try to interact with you as though you’re sane.
It wasn’t a game turtle, nicotinic compounds are well known to reduce symptoms of schizophrenia, because they can’t be patented, you won’t hear much about it. But you get an inkling from the source at wikipedia about schizophrenia and smoking. They didn’t mention in that article that niacin has been used for decades to reduce symptoms of schizophrenia, this is considered QUACKERY, because of BIG PHRMA, not because of the results. That’s the honest truth.
Nicotine is one of the most powerful poisons know to nature.
You will never see a bug on a Nicotinia plant, because it is one of the most effective and devastating insecticides known.
When I was a smoker and a gardener i used to collect up my butts soak them in water and spray the strained liquid over my plants. It was instant death for all Aphids and many other bugs.
A friend used to “companion grow” Nicotinia in his garden as was seldom troubled with insect attacks on nearby plants.
The benefit of niacin was found in a study by a Dr Hoffer(and others) in the 1950s. Since then, the research seems to indicate that it is not effective.
You might be interested in this overview of nicotine :
ncbi.nlm.nih.gov/pubmed/15964073
Google the title and you can see a PDF of the full article. ( Nicotine use in schizophrenia: the self medication hypotheses )
I am particularly interested in this. My brother has schizophrenia and smokes like a metaphorical chimney.
The “conclusions”, whilst interesting are not (ahem) conclusive enough to justify the OP.
“may represent an attempt to self-medicate some of the cognitive deficits”
Is not strong wording.
By the same token, a study that looks into the possible harm of teenagers smoking at the most vulnerable time in their lives for the onset of schizophrenia would be equally welcome, as a substance that interrupts the same sensory gating apparatus for benefits may
just as well do harm when those gating apparatuses are forming in the late teens.
Conclusions
To conclude, there is considerable empirical support for
the idea that smoking in schizophrenia may represent an
attempt to self-medicate some of the cognitive deficits of
this disorder. Proposed are a wide range of beneficial effects
of nicotine which are explained in terms of the drugs’
interaction with dopaminergic as well as glutamatergic
transmitter systems. Given that cigarette smoking lends
itself to titration to achieve specific pharmacological effects,
it is possible that individual patients may use different
dosing strategies to achieve such effects. Combined, such
observations stress the importance of pursuing the development
of nicotinic agonist treatments (Levin and Rezvani,
2002; Martin et al., 2004). Over the last decade attempts
have been made to develop a7 selective agonists such as
DMXBA, 3-(2,4-dimethoxybenzylidene)-anabaseine (Kem,
2000). Such developments appear promising but are yet to
be formally tested for their efficacy in treating impaired
sensory gating, cognitive dysfunctions and negative
symptoms of schizophrenia (Martin et al., 2004). Future
studies exploring the effect of similar substances in
schizophrenia could potentially prove to be influential in
advancing the development of nicotinic agonist treatments
for this disorder. Treatments specifically targeting dysfunctions
in nicotinic–glutamatergic interactions and capable of
effectively treating cognitive deficits in schizophrenia
would be particularly useful. Potentially such a treatment
strategy would not only be a useful advance in treatment of
schizophrenia but also, by providing an alternative to
smoking, reduce the harmful effects, such as elevated risk
for cardiovascular disease (Goff et al., 2005), due to chronic
smoking in this population.